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In light of the most recent evidence, our experimental findings contribute to the growing body of literature highlighting the need for critical re-evaluation of COVID-19 pharmacotherapies in terms of long-term cardiopulmonary safety, especially when used in combination or outside the context of active infection

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Pharmacological activation of NRF2: KEAP1-inhibitors Thus far, the most common class of NRF2-activators mimic the inhibition of NRF2s cytoplasmic negative regulator KEAP1, an adaptor protein for the CUL3/RBX1 E3 ubiquitin ligase complex, to prevent NRF2 proteasomal degradation (Kobayashi et al., 2004)